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Study Identifies Switch That Halts Post‑Stroke Repair, Prolongs Recovery in Mice

The work pinpoints a protein that shuts down microglia’s helpful activity after stroke, offering a clear target for future therapy.

Overview

  • Tokyo University of Science researchers reported in Nature on Wednesday that they found a protein that stops the brain’s repair after stroke.
  • Microglia, the brain’s immune cells, first help healing by releasing growth factors like IGF‑1 but this support fades after about two months.
  • The team identified ZFP384 as the protein that triggers the shutdown of this help in mice and saw the same pattern in human post‑stroke brain tissue.
  • An antisense oligonucleotide that binds Zfp384 messenger RNA kept microglia making IGF‑1 in mice and sustained functional recovery.
  • The researchers said a human version will need redesign, safer delivery, and testing, and they aim for eventual clinical use over the coming decade or two.